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Author:Chiao-Wen Huang, Hong-Ren Yang, Ching-Yi Lin, Sui-Li Hsu, Wen-Chi Ko, and Hui-Fang Ni*
Abstract:
Foot rot is one of emerging diseases for sweet potato in recent years. The objective of this study was to screen fungicides for foot rot diseases caused by Phomopsis destruens. The results showed that mycelial growth was effectively inhibited by prochloraz-manganese, carbendazim + imazalil, iminoctadine triacetate, hexaconazole + thiabendazole, carbendazim + prochloraz, metconazole, carbendazim + hexaconazole and thiabendazole. Spore germination of P. destruens was inhibited by cyprodinil + fludioxonil, prochloraz-manganese, carbendazim + imazalil, iminoctadine triacetate, hexaconazole + thiabendazole, carbendazim + prochloraz, azoxystrobin, iprodione, thiabendazole, metconazole, fluazinam and carbendazim + hexaconazole in 10 mg a.i. L-1. In greenhouse experiments, the results indicated that thiabendazole, hexaconazole + thiabendazole and carbendazim + hexaconazole were effective fungicides for controlling foot rot. Fungicide residues in the case of tubers showed that remains of pesticide in tubers were approximately 0.1 ppm after drenching Thiabendazole after 30 days and 60 days. The results revealed that thiabendazole was low remains of fungicide. Therefore, it is recommended that thiabendazole is the priority chemical for controlling foot rot disease over other fungicides in the future-field experiment of sweet potato.
Key words:Sweet potato (Ipomoea batatas), Foot rot diseases, Phomopsis destruens, Fungicides screening
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